[1]Rani A, Kumar A, Lal A, et al. Cellular mechanisms of cadmiuminduced toxicity: a review[J]. Int J Environ Health Res, 2014, 24(4): 378-399.
[2]Tucovic D, Mirkov I, Kulas J, et al. Dermatotoxicity of oral cadmium is straindependent and related to differences in skin stress response and inflammatory/immune activity[J]. Environ Toxicol Pharmacol, 2020, 75: 103326.
[3]Demenesku J, Popov Aleksandrov A, Mirkov I, et al. Strain differences of cadmiuminduced toxicity in rats: Insight from spleen and lung immune responses[J]. Toxicol Lett, 2016, 256: 33-43.
[4]Xu MY, Wang P, Sun YJ, et al. Joint toxicity of chlorpyrifos and cadmium on the oxidative stress and mitochondrial damage in neuronal cells[J]. Food Chem Toxicol, 2017, 103: 246-252.
[5]Ohrvik H, Ulleras E, Oskarsson A, et al. Effects of cadmium on calcium transporter SPCA, calcium homeostasis and betacasein expression in the murine mammary epithelium[J]. Toxicol Lett, 2011, 201(1): 80-85.
[6]Wang Y, Ji X, Dai S, et al. Cadmium induced redistribution of cholesterol by upregulating ABCA1 and downregulating OSBP[J]. J Inorg Biochem, 2018, 189: 199-207.
[7]Fan T, Chen Y, He Z, et al. Inhibition of ROS/NUPR1dependent autophagy antagonises repeated cadmium exposureinduced oral squamous cell carcinoma cell migration and invasion[J]. Toxicol Lett, 2019, 314: 142-152.
[8]Hambach R, Lison D, D′Haese PC, et al. Coexposure to lead increases the renal response to low levels of cadmium in metallurgy workers[J]. Toxicol Lett, 2013, 222(2): 233-238.
[9]Alver A, Senturk A, Cakirbay H, et al. Carbonic anhydrase Ⅱ autoantibody and oxidative stress in rheumatoid arthritis[J]. Clin Biochem, 2011, 44(17-18): 1385-1389.
[10]Srivastava S, Singh D, Patel S, et al. Role of enzymatic free radical scavengers in management of oxidative stress in autoimmune disorders[J]. Int J Biol Macromol, 2017, 101: 502-517.
[11]Vida C, De Toda IM, Cruces J, et al. Role of macrophages in agerelated oxidative stress and lipofuscin accumulation in mice[J]. Redox Biol, 2017, 12: 423-437.
[12]Bauer ME, Fuente Mde L. The role of oxidative and inflammatory stress and persistent viral infections in immunosenescence[J]. Mech Ageing Dev, 2016, 158: 27-37.
[13]Xu B, Chen S, Luo Y, et al. Calcium signaling is involved in cadmiuminduced neuronal apoptosis via induction of reactive oxygen species and activation of MAPK/mTOR network[J]. PLoS One, 2011, 6(4): e19052.
[14]Lawal AO, Ellis EM. Phospholipase C mediates cadmiumdependent apoptosis in HEK 293 cells[J]. Basic Clin Pharmacol Toxicol, 2012, 110(6): 510-517.
[15]Vaeth M, Zee I, Concepcion AR, et al. Ca2+ signaling but not storeoperated Ca2+ entry is required for the function of macrophages and dendritic cells[J]. J Immunol, 2015, 195(3): 1202-1217.
[16]RosalesCruz P, DomínguezPérez M, ReyesZrate E, et al. Cadmium exposure exacerbates hyperlipidemia in cholesteroloverloaded hepatocytes via autophagy dysregulation[J]. Toxicology, 2018(398-399): 41-51.
[17]Kim JY, Kim SJ, Bae MA, et al. Cadmium exposure exacerbates severe hyperlipidemia and fatty liver changes in zebrafish via impairment of highdensity lipoproteins functionality[J]. Toxicol In Vitro, 2018, 47: 249-258.
[18]Olisekodiaka MJ, Igbeneghu CA, Onuegbu AJ, et al. Lipid, lipoproteins, total antioxidant status and organ changes in rats administered high doses of cadmium chloride[J]. Med Princ Pract, 2012, 21(2): 156-159.
[19]Kidani Y, Elsaesser H, Hock MB, et al. Sterol regulatory elementbinding proteins are essential for the metabolic programming of effector T cells and adaptive immunity[J]. Nat Immunol, 2013, 14(5): 489-499.
[20]Yang W, Bai Y, Xiong Y, et al. Potentiating the antitumour response of CD8+ T cells by modulating cholesterol metabolism[J]. Nature, 2016, 531(7596): 651-655.
[21]Jeong SJ, Lee MN, Oh GT. The role of macrophage lipophagy in reverse cholesterol transport[J]. Endocrinol Metab (Seoul), 2017, 32(1): 41-46.
[22]Zhang L, Xia Q, Zhou Y, et al. Endoplasmic reticulum stress and autophagy contribute to cadmiuminduced cytotoxicity in retinal pigment epithelial cells[J]. Toxicol Lett, 2019, 311: 105-113.
[23]Wang YJ, Yan J, Yin F, et al. Role of autophagy in cadmiuminduced testicular injury[J]. Hum Exp Toxicol, 2017, 36(10): 1039-1048.
[24]Qu KC, Wang ZY, Tang KK, et al. Trehalose suppresses cadmiumactivated Nrf2 signaling pathway to protect against spleen injury[J]. Ecotoxicol Environ Saf, 2019, 181: 224-230.
[25]Jung KT, Oh SH. Polyubiquitinated p62/SQSTM1mediated hemeoxygenase1 stabilization plays a critical role in cadmiuminduced apoptosis of mouse monocyte Raw264.7 cells[J]. Biochem Biophys Res Commun, 2019, 519(2): 409-414.
[26]So KY, Lee BH, Oh SH. The critical role of autophagy in cadmiuminduced immunosuppression regulated by endoplasmic reticulum stressmediated calpain activation in RAW264.7 mouse monocytes[J]. Toxicology, 2018, 393: 15-25.
[27]Gu J, Wang Y, Liu Y, et al. Inhibition of autophagy alleviates cadmiuminduced mouse spleen and human B cells apoptosis[J]. Toxicol Sci, 2019, 170(1): 109-122.
[28]Wang SH, Shih YL, Kuo TC, et al. Cadmium toxicity toward autophagy through ROSactivated GSK3β in mesangial cells[J]. Toxicol Sci, 2009, 108(1): 124-131.
[29]Schulman IG. Liver X receptors link lipid metabolism and inflammation[J]. FEBS Lett, 2017, 591(19): 2978-2991.
[30]Gu J, Dai S, Liu Y, et al. Activation of Ca2+sensing receptor as a protective pathway to reduce Cadmiuminduced cytotoxicity in renal proximal tubular cells[J]. Sci Rep, 2018, 8(1): 1092.
|