[1]Liu M,Liu S,Wang L, et al. Burden of diabetes, hyperglycaemia in China from to 2016: Findings from the 1990 to 2016, global burden of disease study[J]. Diabetes Metab, 2019, 45(3): 286-293.
[2]Sinclair A,Saeedi P,Kaundal A, et al. Diabetes and global ageing among 6599yearold adults: Findings from the International Diabetes Federation Diabetes Atlas, 9thedition[J]. Diabetes Res Clin Pract, 2020, 162: 108078.
[3]Scherer PE,Hill JA. Obesity,diabetes, and cardiovascular diseases: a compendium[J]. Circ Res, 2016, 118(11): 1703-1705.
[4]Haas AV,Mcdonnell ME. Pathogenesis of cardiovascular disease in diabetes[J]. Endocrinol Metab Clin North Am, 2018, 47(1): 51-63.
[5]Sekirov I,Russell SL,Antunes LCM, et al. Gut microbiota in health and disease[J]. Physiol Rev, 2010, 90(3): 859-904.
[6]Jandhyala SM, Talukdar R, Subramanyam C, et al. Role of the normal gut microbiota[J]. World J Gastroenterol, 2015, 21(29): 8787-8803.
[7]Honda K,Littman DR. The microbiota in adaptive immune homeostasis and disease[J]. Nature, 2016, 535(7610): 75-84.
[8]Fung TC, Olson CA, Hsiao EY. Interactions between the microbiota, immune and nervous systems in health and disease[J]. Nat Neurosci, 2017, 20(2): 145-155.
[9]Rooks MG, Garrett WS. Gut microbiota, metabolites and host immunity[J]. Nat Rev Immunol, 2016, 16(6): 341-352.
[10]王安璐,李秋忆,徐浩,等. 肠道菌群与动脉粥样硬化的关系[J]. 中国动脉硬化杂志, 2020, 28(2): 93-98.
[11]Shiny A,Regin B,Mohan V, et al. Coordinated augmentation of NFAT and NOD signaling mediates proliferative VSMC phenotype switch under hyperinsulinemia[J]. Atherosclerosis, 2016, 246: 257-266.
[12]Zetterqvist AV, Berglund LM, Blanco F, et al. Inhibition of nuclear factor of activated Tcells(NFAT)suppresses accelerated atherosclerosis in diabetic mice[J]. PLoS One, 2013, 8(6):e65020.
[13]孙振,李丽华,毛翔,等. 活化T细胞核因子c1在糖尿病血管钙化进展中的作用[J]. 中国动脉硬化杂志, 2020, 28(11): 936-942.
[14]Chen Y,Greenbaum J,Shen H, et al. Association between gut microbiota and bone health: potential mechanisms and prospective[J]. J Clin Endocrinol Metab, 2017, 102(10): 3635-3646.
[15]Yuan T,Yang T,Chen H, et al. New insights into oxidative stress and inflammation during diabetes mellitusaccelerated atherosclerosis[J]. Redox Biol, 2018, 20: 247-260.
[16]Sáncheztapia M,Tovar AR,Torres N. Diet as regulator of gut microbiota and its role in health and disease[J]. Arch Med Res, 2019, 50(5): 259-268.
[17]Li X, Ji Z, Li S, et al.MiR146a5p antagonized AGEsand P.gLPSinduced ABCA1 and ABCG1 dysregulation in macrophages via IRAK1 downregulation[J]. Inflammation, 2015, 38(5): 1761-1768.
[18]Park Y, Pham TX, Lee J. Lipopolysaccharide represses the expression of ATPbinding cassette transporter G1 and scavenger receptor class B, type I in murine macrophages[J]. Inflamm Res, 2012, 61(5): 465-472.
[19]Chen M, Yi L, Zhang Y, et al. Resveratrol attenuates TrimethylamineNOxide(TMAO)induced atherosclerosis by regulating TMAO synthesis and bile acid metabolism via remodeling of the gut microbiota[J]. mBio, 2016, 7(2): e02210-15.
[20]Warrier M,Shih DM,Burrows AC, et al. The TMAOgenerating enzyme flavin monooxygenase 3 is a central regulator of cholesterol balance[J]. Cell Rep, 2015, 10(3): 326-338.
[21]Koren O, Spor A, Felin J, et al. Human oral, gut, and plaque microbiota in patients with atherosclerosis[J]. Proc Natl Acad Sci U S A, 2011, 108(Suppl 1): 4592-4598.
[22]Campbell LA,Kuo C. Chlamydia pneumoniae—an infectious risk factor for atherosclerosis?[J]. Nat Rev Microbiol, 2004, 2(1): 23-32.
[23]Hooper LV,Wong MH,Thelin A, et al. Molecular analysis of commensal hostmicrobial relationships in the intestine[J]. Science, 2001, 291(5505): 881-884.
[24]Turnbaugh PJ,Ley RE,Mahowald MA, et al. An obesityassociated gut microbiome with increased capacity for energy harvest[J]. Nature, 2006, 441(7122): 1027-1031.
[25]Yoshida N,Emoto T,Yamashita T, et al. Bacteroides vulgatus and Bacteroides dorei reduce gut microbial lipopolysaccharide production and inhibit atherosclerosis[J]. Circulation, 2018, 138(22): 2486-2498.
[26]Gu W,Wang Z,Sun Z, et al. Role of NFATc1 in the bonevascular axis calcification paradox[J]. J Cardiovasc Pharmacol, 2020, 75(3): 200-207.
[27]Liu Y,Wang C,Wang G, et al. Loureirin B suppresses RANKLinduced osteoclastogenesis and ovariectomized osteoporosis via attenuating NFATc1 and ROS activities[J]. Theranostics, 2019, 9(3): 4648-4662.
[28]Chen C,Dong B,Wang Y, et al. The role of Bacillus acidophilus in osteoporosis and its roles in proliferation and differentiation[J]. J Clin Lab Anal, 2020, 34(11): e23471.
[29]Belibasakis GN,Emingil G,Saygan B, et al. Gene expression of transcription factor NFATc1 in periodontal diseases[J]. APMIS, 2011, 119(3): 167-172.
[30]Loureno TGB, Spencer SJ, Alm EJ, et al. Defining the gut microbiota in individuals with periodontal diseases: an exploratory study[J]. J Oral Microbiol, 2018, 10(1): 1487741.
[31]Lucas S,Omata Y,Hofmann J, et al. Shortchain fatty acids regulate systemic bone mass and protect from pathological bone loss[J]. Nat Commun, 2018, 9(1): 55.
[32]Sun Y,Wang M,Zhang S, et al. Effects of dachaihu decoction and its “Prescription Elements” on intestinal flora of nonalcoholic fatty liver disease model rats[J]. World J Tradit Chin Med, 2020, 6(1): 97-105.
[33]Ozkul C,Yalinay M,Karakan T. Structural changes in gut microbiome after ramadan fasting: a pilot study[J]. Benef Microbes, 2020, 11(3): 227-233.
[34]Jiao N,Baker SS,Nugent CA, et al. Gut microbiome may contribute to insulin resistance and systemic inflammation in obese rodents: a metaanalysis[J]. Physiol Genomics, 2018(4): 244-254.
|